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Albuterol (salbutamol) is a short-acting β₂-adrenergic receptor agonist studied in biochemistry and pharmacology for its effects on adrenergic receptor mediated signaling pathways. It acts at the molecular level on β₂-adrenergic receptors in bronchial smooth muscles, where its activation leads to modulation of cAMP and downstream mechanisms related with relaxation of such tissue. Low binding to β₁-adrenergic receptors is also observed in receptor selectivity profiling studies.
Albuterol, initially developed through medicinal chemistry optimization during the exploratory pharmaceutical research that occurred in this mid 20th century, has been cited in numerous studies of receptor and signal transduction pharmacology and adrenergic pathway investigation. In scientific research, it has also been applied as the standard reagent for bio-regulating ion responses or mediator release mechanism, or β-adrenergic pathway dynamic investigations under experimentally controlled conditions.
Liquid albuterol for sale at Element CRP is only sold for lab, analytical and educational research purposes. It is not intended for human or animal use in either clinical applications, as treatment supplements, or in products that may be consumed.
From Pubchem
IUPAC Name:4-[2-(tert-butylamino)-1-hydroxyethyl]-2-(hydroxymethyl)phenolSynonyms: Salbutamol, Proventil, VentolineMolecular Formula: C13H21NO3Molecular Weight: 239.31 g/molCAS Number: 18559-94-9PubChem CID: 2083
Albuterol, a short-acting β₂-adrenergic receptor agonist is commonly used in pharmacological studies for the exploration of bronchial smooth muscle signaling dynamics. Stimulation of β₂–receptors activates adenylyl–cyclase mediated cyclic adenosine monophosphate (cAMP) production, which results in subsequent modulation of smooth muscle tone and airway resistance properties under controlled experimental circumstances.
In laboratory and analytical contexts, albuterol has been used to define receptor responsiveness, ligand–receptor affinity and second messenger cascade performance as a function of divergent physiologic stimulation. Formulation-related effects, such as preservative-mediated modulation of bronchial contractility, have been further characterised in preclinical experimental models.
The research studies of albuterol on airway surface liquid homeostasis and on the transport mechanisms for mucociliary clearance. Enhanced stimulation of β(2)-adrenergic receptors has been shown to modulate ion channel function and reprogram epithelial fluid dynamics following experimentally induced changes.
In mechanistic studies, exposure of albuterol has been employed to evaluate airway patency parameters, relaxation kinetics of smooth muscle, and variability in airflow resistance in model respiratory systems. These studies provide new insights into adrenergic control of airway function at the molecular and cellular level.
Albuterol, a β₂-adrenergic agonist, has been investigated as a molecular probe to investigate alveolar epithelial ion transport and fluid reabsorption. In experimental models of the lung, activation of adrenergic signaling pathways modulate sodium channel activity and transepithelial fluid movement.
Clinical trial paradigms investigating time-dependent changes in airway fluid balance have previously used albuterol as a tool to study signaling relations between β2-adrenergic receptors, epithelial transport proteins, and intracellular second messengers.
Beside the most recognized respiratory signaling, albuterol figure as well published in exploratory studies of adrenergic modulation gene transcription and neuro muscular signaling pathways. Its effects have been measured in vitro and molecular biology investigations of transcript expression involved with motor neuron maintenance and neuromuscular junction stability.
These studies concentrate on the intracellular signaling responses, transcriptional regulation, as well as the down-stream protein expression profiles in controlled experimental settings and contribute to a more comprehensive understanding of adrenergic signaling beyond classical airway models.
Albuterol is a well-known β₂-adrenergic receptor agonist widely used in biochemical, pharmacological and signal transduction studies. Its molecular characteristics justify that it can serve as a control compound for the analysis of adrenergic receptor activation, cAMP-dependent signalling cascades and contractile regulatory mechanisms in different experimental conditions.
At Element CRP, liquid albuterol is supplied exclusively for laboratory, analytical, and educational research use. Distribution is restricted to qualified professionals, with no implication of therapeutic application, clinical use, or biological consumption.
Albuterol (salbutamol) is a small-molecule selective beta-2 adrenergic receptor agonist with a phenethanolamine structure that supports preferential ß2-AR engagement over ß1-AR. The compound shows acceptable stability in standard cell culture buffer systems used for adrenergic receptor pharmacology research.
Cell-based research with Albuterol examines ß2-AR agonist activity through Gas/cAMP coupling in airway smooth muscle cell models, bronchial epithelial cell preparations, and receptor-transfected HEK293 cell lines. Standard readouts include cAMP accumulation, downstream PKA-mediated phosphorylation, and ß-arrestin recruitment for biased agonism profiling.
Element CRP supplies this product at 5mg/mL × 30mL. USA-manufactured via chemical synthesis and verified at 99%+ purity by HPLC and Mass Spectrometry. Pre-mixed liquid solution in 30mL or 60mL bottle with dropper; ready for laboratory use. Store at 15-25°C, away from direct light.
WARNING: For research use only. Not for human or animal consumption. Not intended to diagnose, treat, cure, or prevent any disease. For use by qualified research professionals only.
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Element CRP is a chemical supplier. Element CRP is not a compounding pharmacy or chemical compounding facility as defined under 503A of the Federal Food, Drug, and Cosmetic act. Element CRP is not an outsourcing facility as defined under 503B of the Federal Food, Drug, and Cosmetic act.
